Initialising ...
Initialising ...
Initialising ...
Initialising ...
Initialising ...
Initialising ...
Initialising ...
Sato, Katsuya; Onodera, Takefumi*; Omoso, Kota*; Takeda-Yano, Kiyoko*; Katayama, Takeshi*; Ono, Yutaka; Narumi, Issey*
Genome Announcements (Internet), 4(1), p.e01631-15_1 - e01631-15_2, 2016/01
Fujinami, Shun*; Takeda, Kiyoko*; Onodera, Takefumi*; Sato, Katsuya; Shimizu, Tetsu*; Wakabayashi, Yu*; Narumi, Issey*; Nakamura, Akira*; Ito, Masahiro*
Genome Announcements (Internet), 3(5), p.e01005-15_1 - e01005-15_2, 2015/09
Fujinami, Shun*; Takeda, Kiyoko*; Onodera, Takefumi*; Sato, Katsuya; Sano, Motohiko*; Takahashi, Yuka*; Narumi, Issey*; Ito, Masahiro*
Genome Announcements (Internet), 2(5), p.e00866-14_1 - e00866-14_2, 2014/09
Fujinami, Shun*; Takeda, Kiyoko*; Onodera, Takefumi*; Sato, Katsuya; Sano, Motohiko*; Narumi, Issey*; Ito, Masahiro*
Genome Announcements (Internet), 2(3), p.e00458-14_1 - e00458-14_2, 2014/05
Sato, Katsuya; Onodera, Takefumi*; Takeda, Kiyoko; Narumi, Issey*
JAEA-Review 2013-059, JAEA Takasaki Annual Report 2012, P. 110, 2014/03
Nunoshiba, Tatsuo*; Sakata, Yayoi*; Yamauchi, Ayako*; Sato, Katsuya; Onodera, Takefumi*; Narumi, Issey*
JAEA-Review 2013-059, JAEA Takasaki Annual Report 2012, P. 118, 2014/03
Onodera, Takefumi*; Sato, Katsuya; Ota, Toshihiro*; Narumi, Issey*
JAEA-Review 2013-059, JAEA Takasaki Annual Report 2012, P. 111, 2014/03
Fujinami, Shun*; Takeda, Kiyoko; Onodera, Takefumi*; Sato, Katsuya; Sano, Motohiko*; Narumi, Issey*; Ito, Masahiro*
Genome Announcements (Internet), 1(6), P. e01043-13, 2013/12
Onodera, Takefumi; Sato, Katsuya; Ota, Toshihiro*; Narumi, Issei
Extremophiles, 17(1), p.171 - 179, 2013/01
Times Cited Count:10 Percentile:26.66(Biochemistry & Molecular Biology)Onodera, Takefumi; Sato, Katsuya; Ota, Toshihiro*; Narumi, Issei
JAEA-Review 2012-046, JAEA Takasaki Annual Report 2011, P. 104, 2013/01
Nunoshiba, Tatsuo*; Hirasawa, Yukei*; Yamauchi, Ayako*; Sato, Katsuya; Tejima, Kohei*; Onodera, Takefumi; Narumi, Issei
JAEA-Review 2012-046, JAEA Takasaki Annual Report 2011, P. 111, 2013/01
Sato, Katsuya; Tejima, Kohei*; Onodera, Takefumi; Narumi, Issei
JAEA-Review 2012-046, JAEA Takasaki Annual Report 2011, P. 103, 2013/01
Sato, Katsuya; Kikuchi, Masahiro; Ishaque, A. M.*; Oba, Hirofumi*; Yamada, Mitsugu; Tejima, Kohei; Onodera, Takefumi; Narumi, Issei
DNA Repair, 11(4), p.410 - 418, 2012/04
Times Cited Count:24 Percentile:59.51(Genetics & Heredity)In an effort to gain insights into the role of RecFOR proteins in homologous recombination, we generated , and disruptant strains and characterized the disruption effects. Disruption of resulted in severe reduction of the transformation efficiency. On the other hand, the disruptant strain was the most sensitive phenotype to rays, UV irradiation and mitomycin C among the three disruptants. In the disruptant strain, the intracellular level of the LexA1 protein did not decrease following irradiation. These results demonstrate that the RecF protein plays a crucial role in the homologous recombination repair process by facilitating RecA activation. Thus, the RecF and RecR proteins are involved in the RecA activation and the stability of incoming DNA, respectively, during RecA-mediated homologous recombination processes that initiated the ESDSA pathway in .
Sato, Katsuya; Tejima, Kohei; Onodera, Takefumi; Narumi, Issei
JAEA-Review 2011-043, JAEA Takasaki Annual Report 2010, P. 106, 2012/01
Onodera, Takefumi; Sato, Katsuya; Narumi, Issei; Ota, Toshihiro*
JAEA-Review 2011-043, JAEA Takasaki Annual Report 2010, P. 107, 2012/01
Onodera, Takefumi; Narumi, Issei
Nihon Biseibutsu Seitai Gakkai-Shi, 26(2), p.75 - 77, 2011/09
no abstracts in English
Sato, Katsuya; Kikuchi, Masahiro; Oba, Hirofumi*; Yamada, Mitsugu; Tejima, Kohei; Onodera, Takefumi; Narumi, Issei
no journal, ,
In an effort to gain an insight into the role of the RecFOR in DNA repair through homologous recombination in , we generated a RecFOR disruptant strains and characterized the disruption effects. The RecF disruptant strain exhibited sensitivity to DNA-damaging agents, suggesting that RecF has a crucial role in DNA repair. This result indicates that RecF takes an important role in the RecA activation in the homologous recombination process with DNA damage. While, the recombination assay revealed that RecR is essential for the process of homologous intermolecular recombination, suggesting that RecR is important factor in the homologous recombination process without DNA damage. The importance of RecF and RecR in the homologous recombination processes is reversed due to the existence of DNA damage. Therefore, will use the different homologous recombination processes through the RecFOR or RecOR, respectively.
Onodera, Takefumi; Ota, Toshihiro*; Sato, Katsuya; Narumi, Issei
no journal, ,
no abstracts in English
Sato, Katsuya; Tejima, Kohei; Onodera, Takefumi; Narumi, Issei
no journal, ,
no abstracts in English
Nunoshiba, Tatsuo*; Hirasawa, Yukei*; Yamauchi, Ayako*; Sakata, Yayoi*; Aihara, Reiko*; Sato, Katsuya; Tejima, Kohei; Onodera, Takefumi; Narumi, Issei
no journal, ,
no abstracts in English