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Zhou, L.*; Grigoryan, V. L.*; 前川 禎通; Wang, X.*; Xiao, J.*
Physical Review B, 91(4), p.045407_1 - 045407_6, 2015/01
被引用回数：42 パーセンタイル：79.67(Materials Science, Multidisciplinary)The spin Hall and its inverse effects, driven by the spin orbit interaction, provide an interconversion mechanism between spin and charge currents. Since the spin Hall effect generates and manipulates spin current electrically, to achieve a large effect is becoming an important topic in both academia and industries. So far, materials with heavy elements carrying a strong spin orbit interaction, provide the only option. We propose here a new mechanism, using the surface roughness in ultrathin films, to enhance the spin Hall effect without heavy elements. Our analysis based on Cu and Al thin films suggests that surface roughness is capable of driving a spin Hall angle that is comparable to that in bulk Au. We also demonstrate that the spin Hall effect induced by surface roughness subscribes only to the side-jump contribution but not the skew scattering. The paradigm proposed in this paper provides the second, not if only, alternative to generate a sizable spin Hall effect.
Y-family DNA polymerase 
catalyses translesion synthesis and interacts functionally with PCNA2Anderson, H.*; Vonarx, E.*; Pastushok, L.*; 中川 繭; 片渕 淳*; Gruz, P.*; Rubbo, A.*; Grice, D.*; Osmond, M.*; 坂本 綾子; et al.
Plant Journal, 55(6), p.895 - 908, 2008/09
被引用回数：45 パーセンタイル：71.82(Plant Sciences)We assessed the roles of 
and 
in TLS mediated UV resistance. 
defective mutants sensitized growth of roots and whole plants to UV radiation indicating AtPol contributes to UV resistance. alone did not complement the UV sensitivity conferred by deletion of yeast 
, although AtPol exhibited cyclobutane dimer bypass activity and interacted with yeast PCNA in vitro. Co-expression of and , but not 
, restored normal UV resistance and mutation kinetics in the 
mutant. A single residue difference at site 201, which lies adjacent to the lysine ubiquitylated in PCNA, appeared responsible for the inability of PCNA1 to function with AtPol in UV treated yeast. PCNA interacting protein boxes and an ubiquitin-binding motif in AtPol were found to be required for restoration of UV resistance in the mutant by and . These observations indicate AtPol can catalyse TLS past UV induced DNA damage, and link the biological activity of AtPol in UV irradiated cells to PCNA2 and PCNA-and ubiquitin-binding motifs in AtPol.
