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論文

Spontaneous topological Hall effect induced by non-coplanar antiferromagnetic order in intercalated van der Waals materials

高木 寛貴*; 高木 里奈*; 見波 将*; 野本 拓也*; 大石 一城*; 鈴木 通人*; 柳 有起*; 平山 元昭*; Khanh, N.*; 軽部 皓介*; et al.

Nature Physics, 19(7), p.961 - 968, 2023/07

 被引用回数:8 パーセンタイル:96.88(Physics, Multidisciplinary)

In ferromagnets, electric current generally induces a transverse Hall voltage in proportion to the internal magnetization. This effect is frequently used for electrical readout of the spin up and down states. While these properties are usually not expected in antiferromagnets, recent theoretical studies predicted that non-coplanar antiferromagnetic order with finite scalar spin chirality - meaning a solid angle spanned by neighboring spins - can induce a large spontaneous Hall effect even without net magnetization or external magnetic field. This phenomenon, the spontaneous topological Hall effect, can potentially be used for the efficient electrical readout of the antiferromagnetic states, but it has not been experimentally verified due to a lack of appropriate materials hosting such magnetism. Here, we report the discovery of all-in-all-out type non-coplanar antiferromagnetic order in triangular lattice compounds CoTa$$_{3}$$S$$_{6}$$ and CoNb$$_{3}$$S$$_{6}$$. These compounds are reported to host unconventionally large spontaneous Hall effect despite their vanishingly small net magnetization, and our analysis reveals that it can be explained in terms of the topological Hall effect that originates from the fictitious magnetic field associated with scalar spin chirality. These results indicate that the scalar spin chirality mechanism offers a promising route to the realisation of giant spontaneous Hall response even in compensated antiferromagnets, and highlight intercalated van der Waals magnets as a promising quasi-two-dimensional material platform to enable various nontrivial ways of electrical reading and possible writing of non-coplanar antiferromagnetic domains.

口頭

Am含有MOX燃料高線出力試験(B14照射試験),2; ペレット製造及び燃料要素加工

武内 健太郎; 沖田 高敏; 関 正之; 高野 龍雄; 加藤 明文*; 鹿志村 元明

no journal, , 

高速炉サイクルの研究開発ではAm等のマイナーアクチナイド(MA)を数%含有するMOX燃料についての検討が進められている。そこで、MAを含有した高速炉用MOX燃料の熱的性能を確認するため、高速実験炉「常陽」においてAm含有MOX燃料の高線出力での照射試験を実施した。本報告では、照射試験に供した「もんじゅ」仕様を模擬した低密度のMOXペレットの製造及び燃料要素の加工に関して報告する。燃料ペレット製造では、Am含有率の高いPuO$$_{2}$$粉末及び濃縮UO$$_{2}$$粉末を原料粉末とし、均一化混合,ポアフォーマ(密度降下剤)及びバインダの添加混合,造粒,ルブリカントの添加混合,成型,予備焼結及び焼結を行い、燃料ペレットを得た。試験パラメータである燃料ペレットのO/M比及びペレット/被覆管ギャップ幅は、雰囲気制御下での熱処理及び外周研削により、それぞれの試験パラメータの目標値となるように調整した。燃料要素加工では、燃料ペレット及び各部材をSUS316相当ステンレス鋼製被覆管に充填し、タグガス雰囲気を模擬するためにKrを9%混合したHeガス雰囲気中(1気圧)で端栓を溶接して燃料要素を得た。

口頭

${it In vitro}$ screening of target kinases of AtATR

坂本 綾子; 根本 圭一郎*; 関 原明*; 篠崎 一雄*; 澤崎 達也*

no journal, , 

ATR protein is a conserved member of phosphatidylinositol 3-kinase-related kinases involved in cell-cycle checkpoint responses. ATR is activated through DNA damage or stalled replication fork and subsequently phosphorylates downstream cell-cycle components to inhibit the progression of cell cycle. In Arabidopsis, ATR-disrupted mutant shows hypersensitive to DNA damaging treatments or replication inhibitors. However, either upstream or downstream signaling cascade of ATR largely remains unknown in plants. To identify downstream components involved in the ATR-mediated checkpoint responses, we screened Riken Arabidopsis Full-Length (RAFL) cDNA clones by using AlphaScreen luminescence system. Here, the AtATR protein with GST-FLAG tag was synthesized in wheat cell-free extract using synthesized mRNA transcribed ${it in vitro}$, which is bound to protein A-conjugated acceptor beads through anti-FLAG antibody. Similarly, biotin-tagged putative kinases derived from RAFL cDNA clones were synthesized ${it in vitro}$ and bound to streptavidin-coated donor beads. When interaction between AtATR and a putative kinase happens, 520-620 nm emission light from acceptor bead induced by a singlet oxygen from donor bead becomes measurable. After screening of 759 clones, some candidate kinases showed higher luminescence signal than background level, indicating that they have protein-protein interactions with ATR.

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